3 Important Thing For Droping Creatinine

Elevated creatinine is one of the common clinical manifestations of chronic kidney disease in the middle stage of renal impairment. In addition to creatinine, the increase of related toxin indicators such as uric acid and blood urea nitrogen will eventually lead to the occurrence of uremia.

There may be many kidney patients who think this way: "Is it true that uremia will not occur if the creatinine is more than 100, and the risk of uremia will only occur when the creatinine reaches 5 or 600?"

In fact, the level of creatinine mainly reflects the level of endotoxin in our blood, and the increase of creatinine is directly related to the overall damage of renal function. Generally, the more serious the damage, the worse the filtration and metabolism ability, and the corresponding increase in creatinine. The low level of creatinine reflects less damage to renal function, but there is also a risk of developing uremia. When the creatinine level continues to increase, the renal function damage is usually very serious and reaches more than 50%.

Therefore, it does not mean that the increase in creatinine is small, and uremia will not occur, but relatively speaking, the renal function damage is mild and controllable and a reversal is strong, and it can be stabilized and not progressed to a certain extent.

So, whether the creatinine is more than 200 or more than 500, kidney patients should pay attention to the prevention and treatment of uremia, not to say relaxation treatment!

How to stop the damage in time and suppress the continuous increase of creatinine after renal function damage?

If you want to reduce creatinine more effectively, reduce the accumulation of toxins, and significantly reduce the risk of uremia, you must grasp these three points:

1. Timely treatment of lesions of renal activity

The damage of the kidney is said to be the overall decline of renal function when it is larger, and it is a process of successive damage of kidney cells one by one, and then fibrosis and finally the development of sclerosis. Although it is said that damaged kidney cells cannot be regenerated, not all of them are irreversible. In time, the improvement of blood oxygen recovery in the less damaged part can be completely reversed, and some kidney cells can be restored, and kidney function can also be stabilized without continuous decline.

The active lesions of the kidney are mainly the changes of pathological damage, including the proliferation and necrosis of epithelial cells, podocytes, and crescents.

To deal with these damages to kidney cells in a timely manner, it is necessary to stabilize the immune-inflammatory response in the kidneys, improve the blood circulation of the kidneys, and restore the locally damaged kidney cells. Keep the kidneys functioning for a long time without entering the stage of kidney failure. Creatinine does not rise in a short period of time but is more stable.

2 Avoid the causes of elevated creatinine

External triggering factors are also an important aspect of the causes of creatinine changes. The increase in creatinine caused by renal function damage is often a chronic development, and the predisposing factors are mostly acute or random factors.

Conditions such as drugs, dehydration, hypertension, anemia, hypovolemia, and obstruction are all acute and random injury factors, and most of them can be prevented.

However, some require timely medication intervention, such as hypertension, anemia, hypovolemia, obstruction, etc. As long as the cause is treated, the antihypertensive drugs should be taken, the erythropoietin should be corrected, the oxygen-carrying rate of the blood should be improved, and the damaged renal cell sclerosis will be relieved. develop.

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In the course of treatment, the combination of Chinese and Western medicine is used to control, and the recovery effect is better.

3. Strictly control all factors that affect kidney function

The first two points are related to the timely control of the main direct factors that cause the increase of creatinine. In addition, there are some indirect factors that affect both creatinine and direct factors that affect the stability of renal function.

Urinary protein symptoms run through the development of renal function and are an independent risk factor for kidney disease. Although it is said that the loss of urinary protein decreases due to the decrease in filtration after renal function damage, most kidney patients still have different degrees of protein loss after the increase in creatinine. It is still one of the factors that cannot be ignored in accelerating renal failure.

Therefore, to stabilize the condition after the creatinine rises, it is necessary to continue to inhibit protein loss and eliminate the inflammatory response in the kidneys.

Another point is that electrolyte and acid-base metabolism disorders will have a direct impact on creatinine. Electrolytes are mainly due to abnormal metabolism of serum potassium and calcium and phosphorus, which can cause hyperkalemia, hyperphosphatemia, and calcium loss. Acid-base metabolism is prone to cause metabolic acidosis.

The appearance of these symptoms is prone to secondary complications, posing a serious threat to the disease, further aggravating the disease and causing the creatinine index to continue to rise. Therefore, after the creatinine exceeds 265, it is necessary to systematically prevent and treat complications in the later stage of stage 3.

What is your creatinine level now? How long have you had kidney disease?